Sciclone® G3 NGS & NGSx Workstations

Next generation sequencing (NGS) technologies are enabling science at unprecedented rates. As the cost of sequencing declines, throughput continues to increase and the number of applications for which sequencing data can be used is expanding. The Sciclone® G3 NGS and NGSx liquid handling workstations were designed for high throughput, walkaway NGS library preparation. The deck design and intuitive interface virtually eliminate cross contamination and user error. These liquid handlers simplify the construction of up to or greater than 96 libraries per day.

With over 88 automated library prep, sequence capture, and normalization protocols currently developed for use with the Sciclone G3 NGS workstation, the time and effort associated with protocol automation is greatly reduced. Automated library prep protocols have been developed for products from:

  • PerkinElmer
  • Illumina®
  • Agilent®
  • New England BioLabs®
  • 10X Genomics®
  • Roche®
  • Swift Biosciences®
  • Nugen®
  • Pacific Biosystems®
  • Thermo Fisher Scientific®

Our expert team of application scientists continues to collaborate with the scientific and research communities to expand our portfolio of validated methods and develop research applications for future requirements.

Are you looking for a liquid handler for NGS library construction with an on-deck thermal cycler? If so, check out the Sciclone® G3 NGS iQ workstation.

Width1219 mm | 48 in
Depth711 mm | 28 in
For research use only. Not for use in diagnostic procedures.
Advancing Agricultural Genomics with Low-Cost NGS Genotyping

Width: 1219 mm | 48 in
Depth: 711 mm | 28 in

Publications that Cite Using the Sciclone® G3 NGS Workstation

  • Brandstetter, B., Dalwigk, K., Platzer, A., Niederreiter, B., Kartnig, F., Fischer, A., . . . Karonitsch, T. (2019). FOXO3 is involved in the tumor necrosis factor-driven inflammatory response in fibroblast-like synoviocytes. Laboratory Investigation. doi:10.1038/s41374-018-0184-7
  • Corominas, J., Colijn, J. M., Geerlings, M. J., Pauper, M., Bakker, B., Amin, N., . . . Hollander, A. I. (2018). Whole-Exome Sequencing in Age-Related Macular Degeneration Identifies Rare Variants in COL8A1 , a Component of Bruch’s Membrane. Ophthalmology. doi:10.1016/j.ophtha.2018.03.040.
  • Faiz, A., Heijink, I. H., Vermeulen, C. J., Guryev, V., Berge, M. V., Nawijn, M. C., & Pouwels, S. D. (2018). Cigarette smoke exposure decreases CFLAR expression in the bronchial epithelium, augmenting susceptibility for lung epithelial cell death and DAMP release. Scientific Reports,8(1). doi:10.1038/s41598-018-30602-7.
  • Griss, J., Bauer, W., Wagner, C., Maurer-Granofszky, M., Simon, M., Chen, M., . . . Wagner, S. N. (2018). B cells sustain inflammation and improve survival in human melanoma:. doi:10.1101/478735
  • Gösch, L., et al. (2018) A T cell-specific deletion of HDAC1 protects against experimental autoimmune encephalomyelitis. Journal of Autoimmunity. (86).
  • Lange, M., Siemann, S., Woestmann, C., Mcnamara, S., Feldkamp, M., Blueher, A., . . . Rosenthal, A. (2018). Abstract 1413: Automation of a barcoded ctDNA sequencing assay based on hybrid capture on two robotic pipetting platforms enables processing of large numbers of liquid biopsy samples. Cancer Research,78(13 Supplement), 1413-1413. doi:10.1158/1538-7445.am2018-1413.
  • Li, Jin, et al. (2017) Artemisinins Target GABAA Receptor Signaling and Impair a Cell Identity. Cell. 168 (1) 86 – 100.e15.
  • Ong, J., Woldhuis, R. R., Boudewijn, I. M., Berg, A. V., Kluiver, J., Kok, K., . . . Brandsma, C. A. (2019). Age-related gene and miRNA expression changes in airways of healthy individuals. Scientific Reports,9(1). doi:10.1038/s41598-019-39873-0.
  • Rosenberg, A. Z., Wright, C., Fox-Talbot, K., Rajpurohit, A., Williams, C., Porter, C., . . . Halushka, M. K. (2018). XMD-miRNA-seq to generate near in vivo miRNA expression estimates in colon epithelial cells. doi:10.1101/333658.
  • Wong, T. N., Miller, C. A., Jotte, M. R., Bagegni, N., Baty, J. D., Schmidt, A. P., . . . Link, D. C. (2018). Cellular stressors contribute to the expansion of hematopoietic clones of varying leukemic potential. Nature Communications, 9(1). doi:10.1038/s41467-018-02858-0.